Taylor McElroy
Researcher
UAMS
faculty
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Biography and Research Information
OverviewAI-generated summary
Taylor H. McElroy's research investigates the physiological and cognitive impacts of various medical treatments and environmental exposures. Their work has explored the effects of chemotherapy drugs, such as docetaxel, doxorubicin, and cyclophosphamide, on cognitive function and the gut microbiome. Additionally, McElroy has studied the neuroprotective potential of compounds like piperlongumine in mitigating chemotherapy-induced cognitive impairment. Other research areas include the role of mitochondria in neuronal recovery after injury and the cognitive performance of mice exposed to simulated galactic cosmic rays. McElroy's publications also address sex differences in frailty among older adults and iron homeostasis, balancing nutritional needs with health risks in aging populations. With an h-index of 13 and 461 citations across 37 publications, McElroy collaborates with several researchers at the University of Arkansas for Medical Sciences, including Pilar Simmons, Taurean Brown, Fabio Ntagwabira, and Chase Swinton.
Metrics
- h-index: 13
- Publications: 37
- Citations: 468
Selected Publications
- Changes in gut, microbiome, and cognition after doxorubicin, cyclophosphamide, and paclitaxel chemotherapy treatment (2026) DOI
- The adverse effects of chemotherapy on bone mass are not prevented by senolytics (2025) DOI
- Physiological and cognitive changes after treatments of cyclophosphamide, methotrexate, and fluorouracil: implications of the gut microbiome and depressive-like behavior (2023) DOI
- Piperlongumine as a Neuro-Protectant in Chemotherapy Induced Cognitive Impairment (2022) DOI
- Combined Mechanical and Enzymatic Dissociation of Mouse Brain Hippocampal Tissue (2021) DOI
- Combined Mechanical and Enzymatic Dissociation of Mouse Brain Hippocampal Tissue (2021) DOI
- Effects of 16O charged-particle irradiation on cognition, hippocampal morphology and mutagenesis in female mice (2021) DOI
- Cognitive impairment resulting from treatment with docetaxel, doxorubicin, and cyclophosphamide (2021) DOI
Collaboration Network
Top Collaborators
- Sex differences in frailty among older adults
- Comparative toxicities of BPA, BPS, BPF, and TMBPF in the nematode Caenorhabditis elegans and mammalian fibroblast cells
- The role of mitochondria in the recovery of neurons after injury
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
Showing 5 of 12 shared publications
- Comparative toxicities of BPA, BPS, BPF, and TMBPF in the nematode Caenorhabditis elegans and mammalian fibroblast cells
- The role of mitochondria in the recovery of neurons after injury
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Bisphenol TMC disturbs mitochondrial activity and biogenesis, reducing lifespan and healthspan in the nematode <i>Caenorhabditis elegans</i>
Showing 5 of 11 shared publications
- Comparative toxicities of BPA, BPS, BPF, and TMBPF in the nematode Caenorhabditis elegans and mammalian fibroblast cells
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Bisphenol TMC disturbs mitochondrial activity and biogenesis, reducing lifespan and healthspan in the nematode <i>Caenorhabditis elegans</i>
- 6-PPD induces mitochondrial dysfunction and reduces healthspan and lifespan through SKN-1 in Caenorhabditis elegans
Showing 5 of 10 shared publications
- Cognitive impairment resulting from treatment with docetaxel, doxorubicin, and cyclophosphamide
- Piperlongumine as a Neuro-Protectant in Chemotherapy Induced Cognitive Impairment
- Physiological and cognitive changes after treatments of cyclophosphamide, methotrexate, and fluorouracil: implications of the gut microbiome and depressive-like behavior
- Evaluating the effects of low-dose simulated galactic cosmic rays on murine hippocampal-dependent cognitive performance
- Effects of 16O charged-particle irradiation on cognition, hippocampal morphology and mutagenesis in female mice
Showing 5 of 9 shared publications
- Cognitive impairment resulting from treatment with docetaxel, doxorubicin, and cyclophosphamide
- Piperlongumine as a Neuro-Protectant in Chemotherapy Induced Cognitive Impairment
- Physiological and cognitive changes after treatments of cyclophosphamide, methotrexate, and fluorouracil: implications of the gut microbiome and depressive-like behavior
- Evaluating the effects of low-dose simulated galactic cosmic rays on murine hippocampal-dependent cognitive performance
- Combined Mechanical and Enzymatic Dissociation of Mouse Brain Hippocampal Tissue
Showing 5 of 8 shared publications
- Comparative toxicities of BPA, BPS, BPF, and TMBPF in the nematode Caenorhabditis elegans and mammalian fibroblast cells
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Reviewer #1 (Public Review): Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
Showing 5 of 7 shared publications
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- 6-PPD induces mitochondrial dysfunction and reduces healthspan and lifespan through SKN-1 in Caenorhabditis elegans
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Reviewer #1 (Public Review): Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
Showing 5 of 7 shared publications
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Reviewer #1 (Public Review): Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
- Reviewer #3 (Public Review): Mitochondrial stress in GABAergic neurons non-cell autonomously regulates organismal health and aging
Showing 5 of 6 shared publications
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