Rami Ahmad Shahror

Researcher

University of Arkansas for Medical Sciences

faculty

Pharmacology & Toxicology, College of Medicine

10 h-index 24 pubs 410 cited

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Biography and Research Information

OverviewAI-generated summary

Rami Ahmad Shahror's research focuses on the innate immune response, particularly the role of myeloid cells in inflammatory neurodegeneration and ischemic conditions. His work has investigated the mechanisms by which microglial phenotypes are altered in conditions such as brain injury and sepsis, and how these changes contribute to disease progression. Shahror has also explored therapeutic strategies targeting inflammatory pathways, including the investigation of the arginase 1/ornithine decarboxylase pathway and the role of HDAC3 in ameliorating myeloid cell inflammation in retinal ischemic injury. His publications also touch upon advanced techniques for characterizing myeloid cells and potential applications in ocular drug delivery. Shahror collaborates with several researchers at the University of Arkansas for Medical Sciences, including Abdelrahman Y. Fouda, Carol Morris, Esraa Shosha, and Nancy Rusch.

Metrics

  • h-index: 10
  • Publications: 24
  • Citations: 410

Selected Publications

  • Myeloid HDAC3 Deletion Protects Against Traumatic Optic Injury (2025) DOI
  • Disrupting the CD47/SIRPα Axis Protects Against Traumatic Optic Neuropathy (2025) DOI
  • Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology (2024) DOI
  • Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury (2024) DOI
  • Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions (2024) DOI
  • The arginase 1/ornithine decarboxylase pathway suppresses HDAC3 to ameliorate the myeloid cell inflammatory response: implications for retinal ischemic injury (2023) DOI
  • Multi-color Flow Cytometry Protocol to Characterize Myeloid Cells in Mouse Retina Research (2023) DOI

Collaborators

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