Cynthia J.M. Kane

High Impact

Researcher

University of Arkansas for Medical Sciences

faculty

25 h-index 56 pubs 2,394 cited

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Biography and Research Information

OverviewAI-generated summary

Cynthia J.M. Kane's research focuses on the neurobiological effects of ethanol exposure, particularly during critical developmental periods. Her work investigates how ethanol impacts neuroinflammation, myelination, and gene expression in the cerebellum and hippocampus. She has published studies examining these effects in mouse models relevant to fetal alcohol spectrum disorders and alcohol use disorder.

Her recent publications explore the molecular signaling pathways involved in ethanol-induced neuroinflammation, such as the TLR4 and MyD88-dependent pathways. Kane also investigates the long-term consequences of developmental ethanol exposure on neurodevelopment and potential disease onset. Her research group collaborates with several colleagues at the University of Arkansas for Medical Sciences, including Paul D. Drew, James C. Douglas, Tonya Rafferty, and Kalee N. Holloway.

Kane's scholarship metrics include an h-index of 25 and over 2,300 total citations, reflecting a significant body of published work. She is recognized as a highly cited researcher and has been recently active, with publications extending into 2023.

Metrics

  • h-index: 25
  • Publications: 56
  • Citations: 2,394

Selected Publications

  • Ethanol Induces Neuroinflammation in a Chronic Plus Binge Mouse Model of Alcohol Use Disorder via TLR4 and MyD88-Dependent Signaling (2023) DOI
  • Ethanol-induced cerebellar transcriptomic changes in a postnatal model of fetal alcohol spectrum disorders: Focus on disease onset (2023) DOI
  • Cerebellar Transcriptomic Analysis in a Chronic plus Binge Mouse Model of Alcohol Use Disorder Demonstrates Ethanol-Induced Neuroinflammation and Altered Glial Gene Expression (2023) DOI
  • Effects of chronic and binge ethanol administration on mouse cerebellar and hippocampal neuroinflammation (2022) DOI
  • Ethanol effects on cerebellar myelination in a postnatal mouse model of fetal alcohol spectrum disorders (2021) DOI
  • Ethanol modulation of hippocampal neuroinflammation, myelination, and neurodevelopment in a postnatal mouse model of fetal alcohol spectrum disorders (2021) DOI
  • Divergent and overlapping hippocampal and cerebellar transcriptome responses following developmental ethanol exposure during the secondary neurogenic period (2021) DOI
  • Ethanol modulation of cerebellar neuroinflammation in a postnatal mouse model of fetal alcohol spectrum disorders (2021) DOI

Collaborators

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