Prasanth Viswanathan
Researcher
University of Arkansas for Medical Sciences
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Biography and Research Information
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Prasanth Viswanathan's research investigates the molecular mechanisms underlying cancer cell survival and death, with a particular focus on hematological malignancies. His work has explored the role of anti-apoptotic BCL2 family members, such as MCL1, in dictating the selective addiction of Kaposi’s sarcoma-associated herpesvirus-transformed primary effusion lymphoma cell lines. Viswanathan has also studied the mitochondrial ubiquitin ligase MARCHF5 and its cooperative role with MCL1 in inhibiting apoptosis in these cell lines. More recently, his research has extended to the metabolic changes and ferroptosis induction caused by the suppression of TRIP13 in multiple myeloma. Viswanathan has authored four publications and collaborates with researchers at the University of Arkansas for Medical Sciences, including Mark Manzano, Daniel Dunham, and Jackson Gill.
Metrics
- h-index: 1
- Publications: 4
- Citations: 2
Selected Publications
- The Mitochondrial Ubiquitin Ligase MARCHF5 Cooperates with MCL1 to Inhibit Apoptosis in KSHV-Transformed Primary Effusion Lymphoma Cell Lines (2024) DOI
- Suppression of TRIP13 Induces Metabolic Changes and Potentiates Ferroptosis in Multiple Myeloma (2023) DOI
- Expression Ratios of the Antiapoptotic BCL2 Family Members Dictate the Selective Addiction of Kaposi’s Sarcoma-Associated Herpesvirus-Transformed Primary Effusion Lymphoma Cell Lines to MCL1 (2022) DOI
- Expression Ratios of the Anti-Apoptotic BCL2 Family Members Dictate the Selective Addiction of KSHV-Transformed Primary Effusion Lymphoma Cell Lines to MCL1 (2022) DOI
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