Xiaolun Sun Source Confirmed

Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.

High Impact

Assistant Professor

University of Arkansas at Fayetteville

faculty

21 h-index 64 pubs 1,704 cited

Research Areas

Microbial infections and disease research Gut microbiota and health Livestock and Poultry Management Animal Nutrition and Physiology Phytochemicals and Antioxidant Activities Bacterial Genetics and Biotechnology Genomics and Phylogenetic Studies

Links

ORCID Lab Website

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OverviewAI-generated summary

Xiaolun Sun's research focuses on understanding and mitigating infectious diseases, particularly in livestock. His work investigates the role of specific microbial metabolites, such as secondary bile acids, in controlling pathogens like *Clostridium perfringens*, a common cause of necrotic enteritis in chickens. Sun has explored the efficacy of natural compounds like resveratrol in addressing inflammation and vascular dysfunction, as well as the impact of compounds like sodium butyrate on immune responses in chickens to prevent infections like Salmonella.

Further investigations by Sun include the development of vaccines targeting *Clostridium perfringens* sporulation proteins to reduce disease incidence. He also examines the mechanisms by which microbial metabolites can inhibit pathogen virulence, including the role of bile salt hydrolase. Beyond infectious diseases, his recent work has delved into plant genetics, utilizing genome-wide association studies (GWAS) to dissect diverse seed coat patterns in cowpea.

Sun collaborates with several researchers at the University of Arkansas at Fayetteville, including Tahrir Alenezi, Mohit Bansal, Ying Fu, and Rohana Liyanage, with whom he has co-authored multiple publications. His scholarly output includes 64 publications, with a total of 1,704 citations and an h-index of 21, designating him as a highly cited researcher.

Metrics

  • h-index: 21
  • Publications: 64
  • Citations: 1,704

Selected Publications

  • The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid (2025) DOI
  • Identification of Salt Tolerance and Stress Response in US Department of Agriculture Tomato Germplasm at the Seedling Stage (2024) DOI
  • Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence (2024) DOI
  • Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence (2023) DOI
  • Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens (2022) DOI
  • Clostridium perfringens-Induced Necrotic Diseases: An Overview (2022) DOI
  • Triterpenoid CDDO-IM protects against lipopolysaccharide-induced inflammatory response and cytotoxicity in macrophages: The involvement of the NF-κB signaling pathway (2022) DOI
  • Natural Compound Resveratrol Attenuates TNF-Alpha-Induced Vascular Dysfunction in Mice and Human Endothelial Cells: The Involvement of the NF-κB Signaling Pathway (2021) DOI
  • Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization (2021) DOI
  • Specific Secondary Bile Acids Control Chicken Necrotic Enteritis (2021) DOI
  • Sodium butyrate modulates chicken macrophage proteins essential for Salmonella Enteritidis invasion (2021) DOI

Collaborators

Researchers in the database who share publications

Tahrir Alenezi University of Arkansas at Fayetteville 8 shared publications Mohit Bansal University of Arkansas at Fayetteville 5 shared publications Ying Fu University of Arkansas at Fayetteville 5 shared publications Rohana Liyanage University of Arkansas at Fayetteville 4 shared publications Anamika Gupta University of Arkansas at Fayetteville 3 shared publications Bilal Alrubaye University of Arkansas at Fayetteville 3 shared publications Danielle M. Graham University of Arkansas at Fayetteville 2 shared publications Billy M. Hargis University of Arkansas at Fayetteville 2 shared publications

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