Farhana Taher Sumya
Researcher
University of Arkansas for Medical Sciences
unknown
Research Areas
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Biography and Research Information
OverviewAI-generated summary
Farhana Taher Sumya's research focuses on the intricate mechanisms of cellular transport and glycosylation, particularly within the Golgi apparatus. Her work investigates the role of the COG complex, a key component of Golgi trafficking machinery, in glycosylation and its implications for congenital disorders of glycosylation (CDG). Sumya has published studies detailing the impact of COG complex inactivation on Golgi function, the nature of intra-Golgi recycling vesicles, and the development of cellular models for COG complex-related CDG-II diseases. Her publications also include work on rapid COG depletion in mammalian cells using inducible degradation systems and comprehensive proteomic characterization of intra-Golgi trafficking intermediates. Sumya has collaborated extensively with Vladimir Lupashin, Irina D. Pokrovskaya, and Zinia D’Souza at the University of Arkansas for Medical Sciences, with whom she shares multiple publications.
Metrics
- h-index: 4
- Publications: 8
- Citations: 60
Selected Publications
- Deep proteomic profiling of the intra-Golgi trafficking intermediates (2025) DOI
- Comprehensive Proteomic Characterization of the Intra-Golgi Trafficking Intermediates (2024) DOI
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking (2023) DOI
- Rapid COG Depletion in Mammalian Cell by Auxin-Inducible Degradation System (2022) DOI
- Acute COG complex inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra‐Golgi recycling vesicles (2022) DOI
- Acute COG inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra-Golgi recycling vesicles (2022) DOI
- Getting Sugar Coating Right! The Role of the Golgi Trafficking Machinery in Glycosylation (2021) DOI
- Development and Initial Characterization of Cellular Models for COG Complex-Related CDG-II Diseases (2021) DOI
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